Substitution of the carboxyl-terminal domain of apo AI with apo AII sequences restores the potential of HDL to reduce the progression of atherosclerosis in apo E knockout mice.

Lignende værker

• Macrophage apoAI protects against dyslipidemia-induced dermatitis and atherosclerosis without affecting HDL
  af: Tavori, Hagai, et al.
  Udgivet: (2015)
• Current and Emerging Reconstituted HDL-apoA-I and HDL-apoE Approaches to Treat Atherosclerosis
  af: Valanti, Eftaxia-Konstantina, et al.
  Udgivet: (2018)
• Sialidase down-regulation reduces non-HDL cholesterol, inhibits leukocyte transmigration, and attenuates atherosclerosis in ApoE knockout mice
  af: White, Elizabeth J., et al.
  Udgivet: (2018)
• HDL/ApoA-1 infusion and ApoA-1 gene therapy in atherosclerosis
  af: Chyu, Kuang-Yuh, et al.
  Udgivet: (2015)
• HDL-apoA-I Exchange: Rapid Detection and Association with Atherosclerosis
  af: Borja, Mark S., et al.
  Udgivet: (2013)