Exogenous carbon monoxide inhibits neutrophil infiltration in LPS-induced sepsis by interfering with FPR1 via p38 MAPK but not GRK2

समान संसाधन

• Effect of Carbon Monoxide-Releasing Molecules II-liberated CO on Suppressing Inflammatory Response in Sepsis by Interfering with Nuclear Factor Kappa B Activation
  द्वारा: Qin, Weiting, और अन्य
  प्रकाशित: (2013)
• A Novel Role of Exogenous Carbon Monoxide on Protecting Cardiac Function and Improving Survival against Sepsis via Mitochondrial Energetic Metabolism Pathway
  द्वारा: Wang, Xu, और अन्य
  प्रकाशित: (2014)
• Endotoxin promotes neutrophil hierarchical chemotaxis via the p38-membrane receptor pathway
  द्वारा: Wang, Xu, और अन्य
  प्रकाशित: (2016)
• Inhibition of LPS-Induced Activation of Coagulation by p38 MAPK Inhibitor
  द्वारा: Koch, Lutz, और अन्य
  प्रकाशित: (2012)
• New strategy for sepsis: Targeting a key role of platelet-neutrophil interaction
  द्वारा: Wang, Xu, और अन्य
  प्रकाशित: (2014)