Hyperoxia activates ATM independent from mitochondrial ROS and dysfunction

High levels of oxygen (hyperoxia) are often used to treat individuals with respiratory distress, yet prolonged hyperoxia causes mitochondrial dysfunction and excessive reactive oxygen species (ROS) that can damage molecules such as DNA. Ataxia telangiectasia mutated (ATM) kinase is activated by nucl...

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Bibliografiska uppgifter
I publikationen:Redox Biol
Huvudupphovsmän: Resseguie, Emily A., Staversky, Rhonda J., Brookes, Paul S., O’Reilly, Michael A.
Materialtyp: Artigo
Språk:Inglês
Publicerad: Elsevier 2015
Ämnen:
Research Paper
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC4430709/
https://ncbi.nlm.nih.gov/pubmed/25967673
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.redox.2015.04.012
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