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Intracellular ATP does not inhibit Slo2.1 K(+) channels
Under normal physiological conditions, the open probability of Slo2.1 K(+) channels is low. Elevation of cytosolic [Na(+)] and [Cl(−)] caused by ischemia or rapid electrical pacing of cells increases the open probability of Slo2.1 channels and the resulting outward current can stabilize the resting...
Tallennettuna:
| Julkaisussa: | Physiol Rep |
|---|---|
| Päätekijät: | , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Wiley Periodicals, Inc.
2014
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4270230/ https://ncbi.nlm.nih.gov/pubmed/25214519 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.14814/phy2.12118 |
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