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Chlamydial and human heat shock protein 60s activate human vascular endothelium, smooth muscle cells, and macrophages
Both chlamydial and human heat shock protein 60s (HSP 60), which colocalize in human atheroma, may contribute to inflammation during atherogenesis. We tested the hypothesis that chlamydial or human HSP 60 activates human endothelial cells (ECs), smooth muscle cells (SMCs), and monocyte-derived macro...
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Hauptverfasser: | , , , |
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Format: | Artigo |
Sprache: | Inglês |
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American Society for Clinical Investigation
1999
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Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC408102/ https://ncbi.nlm.nih.gov/pubmed/10021466 |
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