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Rapamycin decreases DNA damage accumulation and enhances cell growth of WRN-deficient human fibroblasts
Werner syndrome (WS), caused by mutations at the WRN helicase gene, is a progeroid syndrome characterized by multiple features consistent with accelerated aging. Aberrant double-strand DNA damage repair leads to genomic instability and reduced replicative lifespan of somatic cells. We observed incre...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
BlackWell Publishing Ltd
2014
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4032596/ https://ncbi.nlm.nih.gov/pubmed/24308646 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/acel.12190 |
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