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Prematurity in mice leads to reduction in nephron number, hypertension, and proteinuria
The nephron number at birth is a quantitative trait that correlates inversely with the risk of hypertension and chronic kidney disease later in life. During kidney development, the nephron number is controlled by multiple factors including genetic, epige-netic, and environmental modifiers. Premature...
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| Autores principales: | , , , , , |
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| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
2011
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3896045/ https://ncbi.nlm.nih.gov/pubmed/22243792 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.trsl.2011.10.004 |
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