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Type I interferons down regulate myeloid cell IFNGR by inducing recruitment of an Egr3/Nab1 complex that silences ifngr1 transcription
The ability of type I interferons (IFNs) to increase susceptibility to certain bacterial infections correlates with down regulation of myeloid cell surface IFNGR, the receptor for the type II IFN (IFNγ), and reduced myeloid cell responsiveness to IFNγ. Here, we show that the rapid reductions in mous...
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| Autors principals: | , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2013
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3777655/ https://ncbi.nlm.nih.gov/pubmed/23935197 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1203510 |
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