Receptor guanylyl cyclase B-mediated cGMP signalling controls axon bifurcation of cranial sensory ganglion neurons at the embryonic hindbrain

Παρόμοια τεκμήρια

• Bifurcation of Axons from Cranial Sensory Neurons Is Disabled in the Absence of Npr2-Induced cGMP Signaling
  ανά: Ter-Avetisyan, Gohar, κ.ά.
  Έκδοση: (2014)
• Molecular Analysis of Sensory Axon Branching Unraveled a cGMP-Dependent Signaling Cascade
  ανά: Dumoulin, Alexandre, κ.ά.
  Έκδοση: (2018)
• The receptor guanylyl cyclase Npr2 is essential for sensory axon bifurcation within the spinal cord
  ανά: Schmidt, Hannes, κ.ά.
  Έκδοση: (2007)
• A Short History of cGMP, Guanylyl Cyclases, and cGMP-Dependent Protein Kinases
  ανά: Kots, Alexander Y., κ.ά.
  Έκδοση: (2009)
• Loss of Axon Bifurcation in Mesencephalic Trigeminal Neurons Impairs the Maximal Biting Force in Npr2-Deficient Mice
  ανά: Ter-Avetisyan, Gohar, κ.ά.
  Έκδοση: (2018)