Li, B., Shi, Y., Shu, J., Gao, J., Wu, P., & Tang, S. (2013). Wingless-type Mammary Tumor Virus Integration Site Family, Member 5A (Wnt5a) Regulates Human Immunodeficiency Virus Type 1 (HIV-1) Envelope Glycoprotein 120 (gp120)-induced Expression of Pro-Inflammatory Cytokines via the Ca(2+)/Calmodulin-dependent Protein Kinase II (CaMKII) and c-Jun N-terminal Kinase (JNK) Signaling Pathways. American Society for Biochemistry and Molecular Biology.

Li, Bei, Yuqiang Shi, Jianhong Shu, Junling Gao, Ping Wu, e Shao-Jun Tang. Wingless-type Mammary Tumor Virus Integration Site Family, Member 5A (Wnt5a) Regulates Human Immunodeficiency Virus Type 1 (HIV-1) Envelope Glycoprotein 120 (gp120)-induced Expression of Pro-Inflammatory Cytokines Via the Ca(2+)/Calmodulin-dependent Protein Kinase II (CaMKII) and C-Jun N-terminal Kinase (JNK) Signaling Pathways. American Society for Biochemistry and Molecular Biology, 2013.

Li, Bei, et al. Wingless-type Mammary Tumor Virus Integration Site Family, Member 5A (Wnt5a) Regulates Human Immunodeficiency Virus Type 1 (HIV-1) Envelope Glycoprotein 120 (gp120)-induced Expression of Pro-Inflammatory Cytokines Via the Ca(2+)/Calmodulin-dependent Protein Kinase II (CaMKII) and C-Jun N-terminal Kinase (JNK) Signaling Pathways. American Society for Biochemistry and Molecular Biology, 2013.