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High-intensity interval training increases in vivo oxidative capacity with no effect on P(i)→ATP rate in resting human muscle
Mitochondrial ATP production is vital for meeting cellular energy demand at rest and during periods of high ATP turnover. We hypothesized that high-intensity interval training (HIT) would increase ATP flux in resting muscle (V(Pi→ATP)) in response to a single bout of exercise, whereas changes in the...
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主要な著者: | , , |
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フォーマット: | Artigo |
言語: | Inglês |
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American Physiological Society
2013
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オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3602722/ https://ncbi.nlm.nih.gov/pubmed/23255590 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpregu.00409.2012 |
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