Tai, H., Serrano-Pozo, A., Hashimoto, T., Frosch, M. P., Spires-Jones, T. L., & Hyman, B. T. (2012). The Synaptic Accumulation of Hyperphosphorylated Tau Oligomers in Alzheimer Disease Is Associated With Dysfunction of the Ubiquitin-Proteasome System. American Society for Investigative Pathology.
Citación estilo ChicagoTai, Hwan-Ching, Alberto Serrano-Pozo, Tadafumi Hashimoto, Matthew P. Frosch, Tara L. Spires-Jones, y Bradley T. Hyman. The Synaptic Accumulation of Hyperphosphorylated Tau Oligomers in Alzheimer Disease Is Associated With Dysfunction of the Ubiquitin-Proteasome System. American Society for Investigative Pathology, 2012.
Cita MLATai, Hwan-Ching, et al. The Synaptic Accumulation of Hyperphosphorylated Tau Oligomers in Alzheimer Disease Is Associated With Dysfunction of the Ubiquitin-Proteasome System. American Society for Investigative Pathology, 2012.