Papanicolaou, K. N., Ngoh, G. A., Dabkowski, E. R., O'Connell, K. A., Ribeiro, R. F., Stanley, W. C., & Walsh, K. (2012). Cardiomyocyte deletion of mitofusin-1 leads to mitochondrial fragmentation and improves tolerance to ROS-induced mitochondrial dysfunction and cell death. American Physiological Society.
Stile di citazione ChicagoPapanicolaou, Kyriakos N., Gladys A. Ngoh, Erinne R. Dabkowski, Kelly A. O'Connell, Rogerio F. Ribeiro, William C. Stanley, e Kenneth Walsh. Cardiomyocyte Deletion of Mitofusin-1 Leads to Mitochondrial Fragmentation and Improves Tolerance to ROS-induced Mitochondrial Dysfunction and Cell Death. American Physiological Society, 2012.
Citazione MLAPapanicolaou, Kyriakos N., et al. Cardiomyocyte Deletion of Mitofusin-1 Leads to Mitochondrial Fragmentation and Improves Tolerance to ROS-induced Mitochondrial Dysfunction and Cell Death. American Physiological Society, 2012.