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FXR Regulates Liver Repair after CCl(4)-Induced Toxic Injury

Liver repair is key to resuming homeostasis and preventing fibrogenesis as well as other liver diseases. Farnesoid X receptor (FXR, NR1H4) is an emerging liver metabolic regulator and cell protector. Here we show that FXR is essential to promote liver repair after carbon tetrachloride (CCl(4))-induc...

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Главные авторы: Meng, Zhipeng, Wang, Yandong, Wang, Lin, Jin, Wen, Liu, Nian, Pan, Hao, Liu, Lucy, Wagman, Lawrence, Forman, Barry M., Huang, Wendong
Формат: Artigo
Язык:Inglês
Опубликовано: The Endocrine Society 2010
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Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC3284774/
https://ncbi.nlm.nih.gov/pubmed/20211986
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1210/me.2009-0286
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