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FXR Regulates Liver Repair after CCl(4)-Induced Toxic Injury
Liver repair is key to resuming homeostasis and preventing fibrogenesis as well as other liver diseases. Farnesoid X receptor (FXR, NR1H4) is an emerging liver metabolic regulator and cell protector. Here we show that FXR is essential to promote liver repair after carbon tetrachloride (CCl(4))-induc...
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Hlavní autoři: | , , , , , , , , , |
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Médium: | Artigo |
Jazyk: | Inglês |
Vydáno: |
The Endocrine Society
2010
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Témata: | |
On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3284774/ https://ncbi.nlm.nih.gov/pubmed/20211986 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1210/me.2009-0286 |
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