Ghorpade, D. S., Kaveri, S. V., Bayry, J., & Balaji, K. N. (2011). Cooperative Regulation of NOTCH1 Protein-Phosphatidylinositol 3-Kinase (PI3K) Signaling by NOD1, NOD2, and TLR2 Receptors Renders Enhanced Refractoriness to Transforming Growth Factor-β (TGF-β)- or Cytotoxic T-lymphocyte Antigen 4 (CTLA-4)-mediated Impairment of Human Dendritic Cell Maturation. American Society for Biochemistry and Molecular Biology.
Chicago-stil citatGhorpade, Devram Sampat, Srini V. Kaveri, Jagadeesh Bayry, och Kithiganahalli Narayanaswamy Balaji. Cooperative Regulation of NOTCH1 Protein-Phosphatidylinositol 3-Kinase (PI3K) Signaling By NOD1, NOD2, and TLR2 Receptors Renders Enhanced Refractoriness to Transforming Growth Factor-β (TGF-β)- or Cytotoxic T-lymphocyte Antigen 4 (CTLA-4)-mediated Impairment of Human Dendritic Cell Maturation. American Society for Biochemistry and Molecular Biology, 2011.
MLA-referensGhorpade, Devram Sampat, Srini V. Kaveri, Jagadeesh Bayry, och Kithiganahalli Narayanaswamy Balaji. Cooperative Regulation of NOTCH1 Protein-Phosphatidylinositol 3-Kinase (PI3K) Signaling By NOD1, NOD2, and TLR2 Receptors Renders Enhanced Refractoriness to Transforming Growth Factor-β (TGF-β)- or Cytotoxic T-lymphocyte Antigen 4 (CTLA-4)-mediated Impairment of Human Dendritic Cell Maturation. American Society for Biochemistry and Molecular Biology, 2011.