Männikkö, R., Flanagan, S. E., Sim, X., Segal, D., Hussain, K., Ellard, S., . . . Ashcroft, F. M. (2011). Mutations of the Same Conserved Glutamate Residue in NBD2 of the Sulfonylurea Receptor 1 Subunit of the K(ATP) Channel Can Result in Either Hyperinsulinism or Neonatal Diabetes. American Diabetes Association.

Männikkö, Roope, Sarah E. Flanagan, Xiuli Sim, David Segal, Khalid Hussain, Sian Ellard, Andrew T. Hattersley, ja Frances M. Ashcroft. Mutations of the Same Conserved Glutamate Residue in NBD2 of the Sulfonylurea Receptor 1 Subunit of the K(ATP) Channel Can Result in Either Hyperinsulinism or Neonatal Diabetes. American Diabetes Association, 2011.

Männikkö, Roope, et al. Mutations of the Same Conserved Glutamate Residue in NBD2 of the Sulfonylurea Receptor 1 Subunit of the K(ATP) Channel Can Result in Either Hyperinsulinism or Neonatal Diabetes. American Diabetes Association, 2011.