A mutant Paramecium with a defective calcium-dependent potassium conductance has an altered calmodulin: a nonlethal selective alteration in calmodulin regulation.

The Paramecium mutant, pantophobiac A, has a defect that results in an in vivo loss of calcium-dependent potassium efflux channel activity. This defect is corrected fully by the microinjection of wild-type Paramecium calmodulin into pantophobiac A cells and is partially restored by calmodulins from...

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Bibliographic Details
Main Authors: Schaefer, W H, Hinrichsen, R D, Burgess-Cassler, A, Kung, C, Blair, I A, Watterson, D M
Format: Artigo
Language:Inglês
Published: 1987
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Research Article
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC304990/
https://ncbi.nlm.nih.gov/pubmed/2438688
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