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Altered senescence, apoptosis, and DNA damage response in a mutant p53 model of accelerated aging

The tumor suppressors p16(INK4a) and p53 have been implicated as contributors to age-associated stem cell decline. Key functions of p53 are the induction of cell cycle arrest, senescence, or apoptosis in response to DNA damage. Here, we examine senescence, apoptosis, and DNA damage responses in a mo...

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Hlavní autoři: Hinkal, George W., Gatza, Catherine E., Parikh, Neha, Donehower, Lawrence A.
Médium: Artigo
Jazyk:Inglês
Vydáno: 2009
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2722837/
https://ncbi.nlm.nih.gov/pubmed/19396980
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.mad.2009.01.001
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