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Brief exposure to hyperoxia depletes the glial progenitor pool and impairs functional recovery after hypoxic-ischemic brain injury

Patterns of hypoxic-ischemic brain injury in infants and children suggest vulnerability in regions of white matter development, and injured patients develop defects in myelination resulting in cerebral palsy and motor deficits. Reperfusion exacerbates the oxidative stress that occurs after such inju...

詳細記述

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書誌詳細
主要な著者: Koch, Joshua D, Miles, Darryl K, Gilley, Jennifer A, Yang, Cui-Ping, Kernie, Steven G
フォーマット: Artigo
言語:Inglês
出版事項: 2008
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC2652354/
https://ncbi.nlm.nih.gov/pubmed/18334993
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/jcbfm.2008.15
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