A cyanobacterial LPS antagonist prevents endotoxin shock and blocks sustained TLR4 stimulation required for cytokine expression

Toll-like receptors (TLRs) function as primary sensors that elicit coordinated innate immune defenses through recognition of microbial products and induction of immune and proinflammatory genes. Here we report the identification and biological characterization of a lipopolysaccharide (LPS)-like mole...

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Библиографические подробности
Главные авторы: Macagno, Annalisa, Molteni, Monica, Rinaldi, Andrea, Bertoni, Francesco, Lanzavecchia, Antonio, Rossetti, Carlo, Sallusto, Federica
Формат: Artigo
Язык:Inglês
Опубликовано: The Rockefeller University Press 2006
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Articles
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC2118317/
https://ncbi.nlm.nih.gov/pubmed/16717116
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20060136
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