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Bradykinin potentiation by ACE inhibitors: a matter of metabolism
1. Studies in isolated cells overexpressing ACE and bradykinin type 2 (B(2)) receptors suggest that ACE inhibitors potentiate bradykinin by inhibiting B(2) receptor desensitization, via a mechanism involving protein kinase C (PKC) and phosphatases. Here we investigated, in intact porcine coronary ar...
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| Huvudupphovsmän: | , , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
2002
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1573486/ https://ncbi.nlm.nih.gov/pubmed/12208785 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.bjp.0704862 |
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