Leptin repletion restores depressed β-adrenergic contractility in ob/ob mice independently of cardiac hypertrophy

Impaired leptin signalling in obesity is increasingly implicated in cardiovascular pathophysiology. To explore mechanisms for leptin activity in the heart, we hypothesized that physiological leptin signalling participates in maintaining cardiac β-adrenergic regulation of excitation–contraction coupl...

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Bibliographische Detailangaben
Hauptverfasser: Minhas, Khalid M, Khan, Shakil A, Raju, Shubha VY, Phan, Alexander C, Gonzalez, Daniel R, Skaf, Mike W, Lee, Kwangho, Tejani, Ankit D, Saliaris, Anastasies P, Barouch, Lili A, O'Donnell, Christopher P, Emala, Charles W, Berkowitz, Dan E, Hare, Joshua M
Format: Artigo
Sprache:Inglês
Veröffentlicht: Blackwell Science Inc 2005
Schlagworte:
Cell Physiology
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC1464532/
https://ncbi.nlm.nih.gov/pubmed/15760936
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2005.084566
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