Endothelial nitric oxide synthase is critical for ischemic remodeling, mural cell recruitment, and blood flow reserve

The genetic loss of endothelial-derived nitric oxide synthase (eNOS) in mice impairs vascular endothelial growth factor (VEGF) and ischemia-initiated blood flow recovery resulting in critical limb ischemia. This result may occur through impaired arteriogenesis, angiogenesis, or mobilization of stem...

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Bibliografiska uppgifter
Huvudupphovsmän: Yu, Jun, deMuinck, Ebo D., Zhuang, Zhenwu, Drinane, Mary, Kauser, Katalin, Rubanyi, Gabor M., Qian, Hu Sheng, Murata, Takahisa, Escalante, Bruno, Sessa, William C.
Materialtyp: Artigo
Språk:Inglês
Publicerad: National Academy of Sciences 2005
Ämnen:
Biological Sciences
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC1182413/
https://ncbi.nlm.nih.gov/pubmed/16043715
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0501444102
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