Calmidazolium and arachidonate activate a calcium entry pathway that is distinct from store-operated calcium influx in HeLa cells

Agonists that deplete intracellular Ca(2+) stores also activate Ca(2+) entry, although the mechanism by which store release and Ca(2+) influx are linked is unclear. A potential mechanism involves ‘store-operated channels’ that respond to depletion of the intracellular Ca(2+) pool. Although SOCE (sto...

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Bibliografiska uppgifter
Huvudupphovsmän: Peppiatt, Claire M., Holmes, Anthony M., Seo, Jeong T., Bootman, Martin D., Collins, Tony J., McMDONALD, Fraser, Roderick, H. Llewelyn
Materialtyp: Artigo
Språk:Inglês
Publicerad: Portland Press Ltd. 2004
Ämnen:
Research Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC1133905/
https://ncbi.nlm.nih.gov/pubmed/15130089
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1042/BJ20040097
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