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Exogenous S1P Exposure Potentiates Ischemic Stroke Damage That Is Reduced Possibly by Inhibiting S1P Receptor Signaling

Initial and recurrent stroke produces central nervous system (CNS) damage, involving neuroinflammation. Receptor-mediated S1P signaling can influence neuroinflammation and has been implicated in cerebral ischemia through effects on the immune system. However, S1P-mediated events also occur within th...

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Bibliografske podrobnosti
Main Authors: Eunjung Moon, Jeong Eun Han, Sejin Jeon, Jong Hoon Ryu, Ji Woong Choi, Jerold Chun
Format: Artigo
Jezik:Inglês
Izdano: Hindawi Limited 2015-01-01
Serija:Mediators of Inflammation
Online dostop:http://dx.doi.org/10.1155/2015/492659
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