IL-17A and serum amyloid A are elevated in a cigarette smoke cessation model associated with the persistence of pigmented macrophages, neutrophils and activated NK cells.

While global success in cessation advocacy has seen smoking rates fall in many developed countries, persistent lung inflammation in ex-smokers is an increasingly important clinical problem whose mechanistic basis remains poorly understood. In this study, candidate effector mechanisms were assessed i...

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Bibliografiset tiedot
Päätekijät: Michelle J Hansen, Sheau Pyng J Chan, Shenna Y Langenbach, Lovisa F Dousha, Jessica E Jones, Selcuk Yatmaz, Huei Jiunn Seow, Ross Vlahos, Gary P Anderson, Steven Bozinovski
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Public Library of Science (PLoS) 2014-01-01
Sarja:PLoS ONE
Linkit:http://europepmc.org/articles/PMC4236152?pdf=render
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