Synaptic Reorganization in an Experimental Model of Posttraumatic Epilepsy

Por Dudek, F. Edward, Shao, Li-Rong
Publicado em 2002

Loss of GABAergic Interneurons in Seizure-induced Epileptogenesis

Por Dudek, F. Edward, Shao, Li-Rong
Publicado em 2003

Mossy Fiber Sprouting and Recurrent Excitation: Direct Electrophysiologic Evidence and Potential Implications

Por Dudek, F. Edward, Shao, Li-Rong
Publicado em 2004

Both synaptic and intrinsic mechanisms underlie the different properties of population bursts in the hippocampal CA3 area of immature versus adult rats

Por Shao, Li-Rong, Dudek, F Edward
Publicado em 2009

Repetitive Perforant-Path Stimulation Induces Epileptiform Bursts in Minislices of Dentate Gyrus From Rats With Kainate-Induced Epilepsy

Por Shao, Li-Rong, Dudek, F. Edward
Publicado em 2011

Glycolytic inhibition by 2-deoxy-d-glucose abolishes both neuronal and network bursts in an in vitro seizure model

Por Shao, Li-Rong, Stafstrom, Carl E.
Publicado no J Neurophysiol (2017)

The Glycolytic Metabolite, Fructose-1,6-bisphosphate, Blocks Epileptiform Bursts by Attenuating Voltage-Activated Calcium Currents in Hippocampal Slices

Por Shao, Li-Rong, Wang, Guangxin, Stafstrom, Carl E.
Publicado no Front Cell Neurosci (2018)

Infantile Spasms: An Update on Pre-Clinical Models and EEG Mechanisms

Por Janicot, Remi, Shao, Li-Rong, Stafstrom, Carl E.
Publicado no Children (Basel) (2020)

Glycolytic inhibition: A novel approach toward controlling neuronal excitability and seizures

Por Shao, Li‐Rong, Rho, Jong M., Stafstrom, Carl E.
Publicado no Epilepsia Open (2018)

Pediatric Epilepsy Mechanisms: Expanding the Paradigm of Excitation/Inhibition Imbalance

Por Shao, Li-Rong, Habela, Christa W., Stafstrom, Carl E.
Publicado no Children (Basel) (2019)

2-Deoxyglucose and Beta-Hydroxybutyrate: Metabolic Agents for Seizure Control

Por Rho, Jong M., Shao, Li-Rong, Stafstrom, Carl E.
Publicado no Front Cell Neurosci (2019)

Synaptic and extrasynaptic plasticity in glutamatergic circuits involving dentate granule cells following chronic N-methyl-d-aspartate receptor inhibition

Por He, Shuijin, Shao, Li-Rong, Wang, Yu, Bausch, Suzanne B.
Publicado em 2013

The role of BK-type Ca(2+)-dependent K(+) channels in spike broadening during repetitive firing in rat hippocampal pyramidal cells

Por Shao, Li-Rong, Halvorsrud, Ragnhild, Borg-Graham, Lyle, Storm, Johan F
Publicado em 1999

Increased Kv1 Channel Expression May Contribute to Decreased sIPSC Frequency Following Chronic Inhibition of NR2B-Containing NMDAR

Por He, Shuijin, Shao, Li-Rong, Rittase, W Bradley, Bausch, Suzanne B
Publicado em 2012

Reduced K(+) Channel Inactivation, Spike Broadening, and After-Hyperpolarization in Kvβ1.1-Deficient Mice with Impaired Learning

Por Giese, Karl Peter, Storm, Johan F., Reuter, Dirk, Fedorov, Nikolai B., Shao, Li-Rong, Leicher, Thorsten, Pongs, Olaf, Silva, Alcino J.
Publicado em 1998

Presynaptic Ca(2+)-Activated K(+) Channels in Glutamatergic Hippocampal Terminals and Their Role in Spike Repolarization and Regulation of Transmitter Release

Por Hu, Hua, Shao, Li-Rong, Chavoshy, Sorush, Gu, Ning, Trieb, Maria, Behrens, Ralf, Laake, Petter, Pongs, Olaf, Knaus, Hans Günther, Ottersen, Ole Petter, Storm, Johan F.
Publicado no J Neurosci (2001)